Development of T-leukaemias in CD45 tyrosine phosphatase-deficient mutant lck mice.

نویسندگان

  • M Baker
  • J Gamble
  • R Tooze
  • D Higgins
  • F T Yang
  • P C O'Brien
  • N Coleman
  • S Pingel
  • M Turner
  • D R Alexander
چکیده

The CD45 tyrosine phosphatase lowers T-cell antigen receptor signalling thresholds by its positive actions on p56(lck) tyrosine kinase function. We now show that mice expressing active lck(F505) at non-oncogenic levels develop aggressive thymic lymphomas on a CD45(-/-) background. CD45 suppresses the tumorigenic potential of the kinase by dephosphorylation of the Tyr394 autophosphorylation site. In CD45(-/-) thymocytes the kinase is switched to a hyperactive oncogenic state, resulting in increased resistance to apoptosis. Transformation occurs in early CD4(-)CD8(-) thymocytes during the process of TCR-beta chain rearrangement by a recombinase-independent mechanism. Our findings represent the first example in which a tyrosine phosphatase in situ prevents the oncogenic actions of a SRC: family tyrosine kinase.

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عنوان ژورنال:
  • The EMBO journal

دوره 19 17  شماره 

صفحات  -

تاریخ انتشار 2000